The human wrist exhibits adaptive stiffness modulability: joint stiffness anisotropy can be actively regulated through muscle co-contraction. This functionality is essential for stable manipulation, yet the underlying morphological factors remain unclear. To identify these factors, we developed an anatomically accurate anthropomimetic soft robotic forearm comprising eight independently movable carpal bones interconnected by ligaments, 22 actuated muscles, and compliant fingertips. We measured wrist joint stiffness under four muscle activation patterns across three skeletal configurations: anatomically normal carpal bones, a fused proximal carpal row, and a geometric ellipsoidal skeleton. The stiffness ellipse exhibited low stiffness along the dart-throwing motion (DTM) direction when finger muscles were activated, but high stiffness along the same direction when wrist and finger muscles were activated simultaneously. These results agree with previously reported human measurements, demonstrating that precise anatomical replication reproduces human-like stiffness modulability. Fusing the proximal carpal row eliminated the low DTM-direction stiffness under finger muscle activation, while the geometric ellipsoidal skeleton showed poor stiffness ellipse reorientation across all conditions. Carpal bone motion analysis revealed significantly opposing coupling patterns between wrist and finger muscles at the proximal carpal row, accompanied by a consistent but non-significant trend at the midcarpal joint, providing a mechanical explanation for this modulation. These findings demonstrate that carpal bone morphology plays a dominant role in human wrist stiffness modulation and provide design principles for humanoid robot wrists.

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